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Hypovolemic Shock Case Study

John was involved in a car accident. He was rushed to the nearest emergency department as he is bleeding profusely. Upon arrival, his blood pressure has dropped, his heart rate and respirations are rapid, and his skin is cold and clammy. His output is decreased and he is disoriented. John is suffering from hypovolemic shock due to active fluid loss



Hypovolemic shock is one of the most common cardiac complications.


The pathophysiology of hypovolemic shock include the following processes:

  1. Fluid loss. Fluid loss can either be internal or external fluid loss.
  2. Compensatory mechanism.The resulting drop in the arterial blood pressure activates the body’s compensatory mechanisms in an attempt to increase the body’s intravascular volume.
  3. Venous return. Diminished venous return occurs as a result of the decrease in arterial blood pressure.
  4. Preload. The preload or the filling pressure becomes reduced.
  5. Stroke volume. The stroke volume is decreased.
  6. Cardiac output.Cardiac output is decreased because of the decrease in stroke volume.
  7. Arterial pressure.Reduced mean arterial pressure follows as the cardiac output gradually decreases.
  8. Compromised cell nutrients. As the tissue perfusion decreases, the delivery of nutrients and oxygen to the cells are decreased, which could ultimately lead to multiple organ dysfunction syndrome.


Hypovolemic shock usually results from acute blood loss- about one-fifth of the total volume.

  • Internal fluid loss. Internal fluid losses can result from hemorrhage or third-space fluid shifting.
  • External fluid loss. External fluid loss can result from severe bleeding or from severe diarrhea, diuresis, or vomiting.
  • Inadequate vascular volume. Inadequate vascular volume leads to decreased venous return and cardiac output.

Clinical Manifestations

Hypovolemic shock requires early recognition of signs and symptoms.

  • Hypotension. Hypovolemic shock produces hypotension with narrowed pulse pressure.
  • Cognitive. The patient experiences decreased sensorium.
  • Tachycardia. The body compensates for the decreased cardiac output by pumping faster than normal, resulting in tachycardia.
  • Rapid, shallow respirations. Due to the decrease in oxygen delivery around the body systems, the respiratory system compensates by rapid, shallow respirations.
  • Oliguria. There is oliguria or decreased urine output of less than 25ml/hour.
  • Clammy skin. The patient develops cool, clammy, and pale skin.


For prevention of hypovolemic shock, the following must be implemented:

  • Early detection. Recognize patients with conditions that reduce blood volume as at-risk patients.
  • Accurate I&O. Estimate fluid loss and replace, as necessary, to prevent hypovolemic shock.


Hypovolemic shock, if left untreated, would result to the following complications:

  • Acute respiratory distress syndrome.Acute respiratory distress syndrome occurs when fluid builds up in the tiny, elastic air sacs in the lungs.
  • Acute tubular necrosis. Acute tubular necrosis is a kidney disorder involving damage to the tubule cells of the kidneys, which can lead to acute kidney failure.
  • Disseminated intravascular coagulation. Disseminated intravascular coagulation is a pathological process characterized by a widespread activation of the clotting cascades that results in the formation of blood clots in the small blood vessels.
  • Multiple organ dysfunction syndrome. Multiple organ dysfunction syndrome is the end result of hypovolemic shock.

Assessment and Diagnostic Findings

No single symptom or diagnostic test establishes the diagnosis or severity of shock.

  • Laboratory findings. There is elevated potassium, serum lactate, and blood urea nitrogen levels.
  • Urine characteristics.The urine specific gravity and urine osmolality are increased.
  • Blood considerations. Decreased blood pH, partial pressure of oxygen, and increased partial pressure of carbon dioxide.

Medical Management

Emergency treatment measures must include prompt and adequate fluid and blood replacement to restore intravascular volume and raise blood pressure.

  • Volume expansion. Saline solution or lactated Ringer’s solution, then possibly plasma proteins or other plasma expanders, may produce adequate volume expansion until whole blood can be matched.
  • Pneumatic antishock garment. A pneumatic antishock garment counteracts bleeding and hypovolemia by slowing or stopping arterial bleeding; by forcing any available blood from the lower body to the brain, heart, and other vital organs; and by preventing return of the available circulating blood volume to the legs.
  • Treat underlying cause. If the patient is hemorrhaging, efforts are made to stop the bleeding or if the cause is diarrhea or vomiting, medications to treat diarrhea and vomiting are administered.
  • Redistribution of fluid. Positioning the patient properly assists fluid redistribution, wherein a modified Trendelenburg position is recommended in hypovolemic shock.

Pharmacologic Therapy

If fluid administration fails to reverse hypovolemic shock, the following are given:

  • Vasoactive drugs. Vasoactive drugs that prevent cardiac failure are given.
  • Insulin is administered if dehydration is secondary to hyperglycemia.
  • Desmopressin (DDAVP). Desmopressin is administered for diabetes insipidus.
  • Antidiarrheal drugs. If dehydration is due to diarrhea, antidiarrheal medications are administered.
  • Antiemetics. If the cause of diarrhea is vomiting, antiemetics are given.

Nursing Management

Primary prevention of shock is an essential focus of nursing care.

Nursing Assessment

Assessment of the following is vital in hypovolemic shock:

  • History. The history is vital in determining the possible causes and in determining the work-up.
  • Vital signs. Vital signs, prior to arrival at the emergency department, should also be noted.
  • Trauma. In patients with trauma, determine the mechanism of injury and any information that may heighten suspicion of certain injuries.

Nursing Diagnosis

Based on the assessment data, the major nursing diagnoses are:

Nursing Care Planning & Goals

Main Article:4 Hypovolemic Shock Nursing Care Plans

The major goals for the patient are:

  • Maintain fluid volume at a functional level.
  • Report understanding of the causative factors of fluid volume deficit.
  • Maintain normal blood pressure, temperature, and pulse.
  • Maintain elastic skin turgor, most tongue and mucous membranes, and orientation to person, place, and time.

Nursing Interventions

Nursing care focuses on assisting with treatment targeted at the cause of the shock and restoring intravascular volume.

  • Safe administration of blood. It is important to acquire blood specimens quickly, to obtain baseline complete blood count, and to type and crossmatch the blood in anticipation of blood transfusions.
  • Safe administration of fluids. The nurse should monitor the patient closely for cardiovascular overload, signs of difficulty of breathing, pulmonary edema, jugular vein distention, and laboratory results.
  • Monitor weight. Monitor daily weight for sudden decreases, especially in the presence of decreasing urine output or active fluid loss.
  • Monitor vital signs. Monitor vital signs of patients with deficient fluid volume every 15 minutes to 1 hour for the unstable patient, and every 4 hours for the stable patient.
  • Oxygen administration. Oxygen is administered to increase the amount of oxygen carried by available hemoglobin in the blood.


Expected outcomes for the patient include:

  • Maintained fluid volume at a functional level.
  • Reported understanding of the causative factors of fluid volume deficit.
  • Maintained normal blood pressure, temperature, and pulse.
  • Maintained elastic skin turgor, most tongue and mucous membranes, and orientation to person, place, and time.

Documentation Guidelines

The focus of documentation include:

  • Degree of deficit and current sources of fluid intake.
  • I&O, fluid balance, changes in weight, presence of edema, urine specific gravity, and vital signs.
  • Results of diagnostic studies.
  • Functional level and specifics of limitations.
  • Needed resources and adaptive devices.
  • Availability and use of community resources.
  • Plan of care.
  • Teaching plan.
  • Client’s responses to interventions, teachings, and actions performed
  • Attainment or progress towards desired outcomes.
  • Modifications to plan of care.

Practice Quiz: Hypovolemic Shock

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Practice Quiz: Hypovolemic Shock

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Chief Complaint: 31-year-old African American male injured while rock-climbing.

History of Present Illness: This 31-year-old male was rock-climbing with two friends at a national park 14 miles away from the nearest hospital when he suddenly lost his footing and slid 18 feet to the ground. Both friends who witnessed the fall said that he slid against sharp rock all of the way down, landing almost in a standing-up position, finally slumping to the ground. They also said that his head was not jarred during the slide. The man was alert and oriented when his friends reached him, and could move all four extremities quite easily. He had multiple scrapes over his anterior torso and a large gash over his right anterior upper thigh (near the groin) which was bleeding profusely. A makeshift tourniquet slowed the bleeding. The party was able to radio to the park ranger's station for help. The ranger, in turn, contacted the "med-evac" unit at the city hospital. The helicopter located and evacuated the man. A large-bore IV was placed in each arm in-flight, and normal saline fluid was administered intravenously. The patient became increasingly disoriented during the flight, reaching the emergency room about 40 minutes after the fall.

Past Medical History: According to the man's friends, the patient was in good health prior to the accident, with no history of allergies, anemia, bleeding disorders, or diabetes mellitus.

Family History: Family history was not attainable. The patient's family lived in another state and could not be contacted.

Physical Examination: The patient was lethargic but responsive to shouting and sternal pinch. He had multiple abrasions over his chin, neck, anterior thorax, and abdomen. A six-inch-long, half-inch deep laceration was noted in the right inguinal region, extending into the right, upper thigh. The tourniquet placed in this area was soaked with blood. Height 6' 2", weight 205 lbs. Vital signs were as follows: HR = 112 (supine) and 128 (sitting), BP = 108 / 60 (supine) and 92 / 52 (sitting), RR = 32, rectal temp = 99.4oF. Skin was cold and clammy, and nail beds, palms, and mucous membranes were pale. Carotid, radial, left femoral, and dorsalis pedis pulses were all weak and thready. Cranial nerves, to the extent that they could be tested, were intact bilaterally. Pupils were equal, regular, and reactive to light. External jugular venous collapse point was not visible in either the sitting or recumbent position. Heart sounds were regular, tachycardic, with no murmur, S3 or S4 sounds. Lungs were clear to percussion and auscultation. Abdominal guarding was noted, attributable to multiple lacerations; no masses were felt. Blood was drawn, typed, and crossed. A urethral catheter was placed to monitor urinary output, and another catheter was placed into the right subclavian vein and threaded into the superior vena cava to monitor central venous pressure. A cardiovascular surgeon was consulted for repair of the lacerated right femoral artery.

Laboratory Studies: Laboratory studies of the venous blood revealed the following:

Blood Type A+                 = 
Total white blood cell (WBC) count = 7,400 WBCs /mm3(normal = 4,000 to 11,000)
Differential WBC count revealed 59% neutrophils (normal = 55-70%)
Hematocrit = 46% (normal = 42-54%)
Hemoglobin = 15.0 gm / dl (normal = 14-18 gm / dl)
Sodium (Na+) = 138 mEq / L (normal = 136-145 mEq / L)
Potassium (K+) = 5.1 mEq / L (normal = 3.5-5.1 mEq / L)
Chloride (Cl-) = 104 mEq / L (normal = 96-106 mEq / L)
BUN = 27 mg / dl(normal = 6 - 23 mg / dl)
Creatinine = 1.9 mg / dl(normal = 0.7 - 1.5 mg / dl)
Glucose = 165 mg / dl(normal = 70 - 160 mg / dl)
SGPT = 41 IU / L(normal = 0-33 IU / L)
SGOT = 48 IU / L(normal = 0 41 IU / L)

Laboratory studies of the arterial blood revealed the following:

Blood pH = 7.28 (normal = 7.35-7.45)
pCO2 = 31 mm Hg(normal = 40 mm Hg)
pO2 = 78 mm Hg(normal = 90-100 mm Hg)
Hemoglobin - O2 saturation = 88% (normal = 94-100%)
[HCO3-] = 14 mEq / L(normal = 22-26 mEq / L)

Urinary output in first 60 minutes in ER was 20 ml (color was dark yellow).
Urine specific gravity = 1.029 (normal = 1.003 - 1.030).
Central venous pressure ranged from 1 to 3 cm H2O throughout the cardiac cycle
(normal = range = 5.5 to 13 cm H2O).

ECG revealed normal sinus rhythm with slight ST-depression in most leads.

As soon as whole blood became available, 2 units were rapidly transfused into this patient.


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